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◆ American Journal of Physiology-Heart and Circulatory Physiology2026-01-07· Viral Myocarditis

Cardiomyocyte β-arrestins mediate inflammation and cGAS-STING activation in CVB3 viral myocarditis

Emilio Y. Lucero, Haoran Jiang, Vincent M. D’Anniballe, Natalia Pakharukova, Daniella Galtes, Dana K. Bassford, Arun Jyothidasan, Kirk U. Knowlton, Michael D. Gunn, R J Lefkowitz, Howard A. Rockman

原始摘要(英文原文)· Original abstract
Viral myocarditis can lead to dilated cardiomyopathy in adults, yet the molecular mechanisms that induce cardiac inflammation in viral myocarditis remain unclear. β-Arrestins are scaffolding proteins that mediate intracellular signaling during viral infection. We demonstrated that cardiomyocyte β-arrestins are necessary for immune cell recruitment, induction of cardiac apoptosis, and cGAS-STING pathway activation during acute coxsackievirus B3 infection. Targeting β-arrestins appears to decrease the inflammatory response in acute viral myocarditis.
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Cardiomyocyte β-arrestins mediate inflammation and cGAS-STING activation in CVB3 viral myocarditis — 科研速览 Science Skim