Eojina Lee, Soyoung Ko, Chul Chang
Background and Objectives: Delayed contour irregularities may become apparent years after otherwise successful autologous rhinoplasty, particularly in patients with thin nasal skin. Thin-skinned rhinoplasty, autologous graft behavior, and nasal aging have been studied individually, but their potential interaction remains unclear. This review examines current evidence on age-related changes in the nasal soft-tissue envelope, thin nasal skin, autologous graft behavior, and their potential effects on long-term rhinoplasty outcomes. Materials and Methods: A structured narrative review was conducted using PubMed as the primary database, with supplementary searches of Google Scholar and reference lists. Four predefined search domains addressed thin nasal skin, nasal aging, autologous graft behavior, and preservation and camouflage techniques. The PubMed searches identified 463 records. After duplicate removal and title and abstract screening, 205 publications were retained for further assessment. Additional relevant publications were identified through supplementary searches. Evidence was synthesized qualitatively because of differences in study design, surgical technique, follow-up duration, and outcome assessment. Results: Thin nasal skin is associated with increased visibility of underlying structural features after rhinoplasty. Aging is associated with changes in skin thickness and elasticity, soft-tissue volume, and the underlying nasal framework. These findings provide a possible basis for changes in soft-tissue camouflage over time, but direct longitudinal evidence linking aging to delayed graft visibility is lacking. Late contour changes may also result from graft warping or resorption, scar remodeling, or changes in the native nasal framework. The ability of preservation and camouflage techniques to prevent aging-related delayed contour visibility has not been established. Conclusions: Age-related changes in the nasal soft-tissue envelope may contribute to delayed contour visibility after rhinoplasty. However, direct longitudinal evidence showing that these changes increase the visibility of an otherwise stable autologous graft is lacking. This relationship should therefore be considered a hypothesis rather than an established causal mechanism. Prospective longitudinal studies using objective measurements of soft-tissue thickness, graft morphology, and external nasal contour are needed to test this hypothesis.