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◆ International journal of radiation oncology, biology, physics2026-09-25

Memantine Prevents Radiation-Induced Cognitive Decline in Brain Metastases - A Randomized Placebo-Controlled Trial.

Haripriya Parapparambil Surendran, Debnarayan Dutta, Kalavagunta Sruthi, Mazhuvancherry Kesavan Unnikrishnan, Dhanya Chandran, Renjitha Bhaskaran, Narmadha Mukunthu Poornachary, Parasuraman Ayiramuthu, Sabitha Mangalathillam, Ajay Sasidharan, Anoop Remesan Nair, Pushpaja Kuttassery Ullattil, Haridas M Nair, Megha Philip, Annex Haridas, Vidya Nottamkandath, Sheejamol V S, Rajesh Kannan, Wesley Mannirathil Jose, Keechilat Pavithran, Nikhil Krishna Haridas, Sourabh Radhakrishnan, Rakesh M P, Sajesh Menon, Suhas Udayakumaran, Sreehari N R, Dalvin Thomas

一句话结论 · In one sentence

Memantine preserved cognitive function relative to placebo and improved QOL in patients receiving radiotherapy for BM, with benefits observed in both SRS and HA/WBRT cohorts.

原始摘要(英文原文)· Original abstract
BACKGROUND: This prospective, double-blind, placebo-controlled, randomized clinical trial evaluated the role of memantine in preventing cognitive decline following radiotherapy (RT) for brain metastases (BM). METHODS: Patients with BM undergoing either stereotactic radiosurgery (SRS) or whole-brain radiotherapy with or without hippocampal avoidance (HA/WBRT) were randomized to receive memantine (20 mg/day) or placebo for 6 months post-RT. Primary outcomes included cognitive function (Addenbrooke's Cognitive Examination [ACE]), quality of life (QoL; EORTC QLQ-C30 and BN20), white matter volume changes (MRI T2 FLAIR), plasma memantine levels (LC-MS/MS), and adverse events (CTCAE v5.0). RESULTS: 130 patients were enrolled (memantine n=66; placebo n=64; SRS n=75; HA/WBRT n=55). At 6 months post-RT, cognitive decline was significantly higher in the placebo group (mean ACE: 72.9 ± 20.2) than in the memantine group (83.9 ± 10.8; p=0.005). Memantine improved memory (p<0.001), delayed recall (p<0.001), and verbal fluency (p=0.007), with better domain-specific cognitive preservation in the SRS subgroup (p<0.001). Memantine was associated with improved cognitive function in both HA/WBRT and SRS cohorts; the effect was statistically significant in HA/WBRT patients, whereas in the SRS subgroup better domain-specific cognitive preservation was observed but the global cognitive effect estimate was underpowered. Global health status improved with memantine (median change +33.3 points) but was unchanged with placebo (between-arm difference +33.3 points, 95% CI +16.7 to +41.7; p<0.001). Common toxicities included appetite loss (25.7% vs. 12.5%) and gastric irritation (7.5% vs. 0%). White matter volume correlated negatively with cognitive decline (r = -0.544; p = 0.055). The optimal trough concentration (118.06 ng/mL) was achieved at 5 mg BID; while levels exceeded the alert threshold (300 ng/mL) at 10 mg BID for 6.25% patients. CONCLUSION: Memantine preserved cognitive function relative to placebo and improved QOL in patients receiving radiotherapy for BM, with benefits observed in both SRS and HA/WBRT cohorts.
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Memantine Prevents Radiation-Induced Cognitive Decline in Brain Metastases - A Randomized Placebo-Controlled Trial. — 科研速览 Science Skim